Purbali A, Trinath J, Sajeli A, Yogeeswari P. Combining Structure-based and 3D QSAR Pharmacophore Models to Discover Diverse Ligands against EGFR in Oral Cancer. Future Medicinal Chemistry. (Accepted, January 2022).
Eugene SP, Trinath J*. Endoplasmic Reticulum Stress in Intestinal Inflammation: Implications of Bile Acids. Accepted, PINSA, India. 2021 *Corresponding author.
V. Sudhakar Reddy, Trinath Jamma, and G. Bhanuprakash Reddy. Small Heat Shock Proteins in Inflammatory Diseases. Book Chapter Accepted. Heat Shock Protein Journal.
Eugene SP, Reddy VS, Trinath J*. Endoplasmic Reticulum Stress and Intestinal Inflammation: A Perilous Union. Front Immunol. 2020; 11:543022. * Corresponding author.
Hang S, Paik D, Yao L, Kim E, Jamma T, Lu J, Ha S, Nelson BN, Kelly SP, Wu L, Zheng Y, Longman RS, Rastinejad F, Devlin AS, Krout MR, Fischbach MA, Littman DR, Huh JR. Bile acid metabolites control Th17 and Treg cell differentiation. Nature. 2019;576(7785):143-148.1.
Reddy VS, Trinath J, Reddy GB. Implications of homocysteine in protein quality and control processes. Biochimie. 2019;165:19-31.
Trinath J, Bayry J. Current trends with FOXP3+ regulatory T cell immunotherapy to contest autoimmunity and inflammation. Immunotherapy. 2019;11(9):755-758.
Reddy VS, Madala SK, Trinath J, Reddy GB. Extracellular small heat shock proteins: exosomal biogenesis and function. Cell Stress Chaperones. 2018;23(3):441-454.
Before Joining BITS:
Mohan S Maddur, Emmanuel Stephen-Victor, Mrinmoy Das, Praveen Prakhar, Varun K Sharma, Vikas Singh, Magalie Rabin, Trinath J, Kithiganahalli N Balaji, Francis Bolgert, Jean-Michel Vallat, Laurent Magy, Srini V Kaveri, Jagadeesh Bayry. (2017). Regulatory T cell frequency, but not plasma IL-33 levels represents potential immunological biomarker to predict clinical response to intravenous immunoglobulin therapy. J Neroinflammation. 14(1):58.
Maddur MS, Trinath J, Rabin M, Bolgert F, Guy M, Vallat JM, Magy L, Balaji KN, Kaveri SV, Bayry J. (2015). Intravenous immunoglobulin-mediated expansion of regulatory T cells in autoimmune patients is associated with increased prostaglandin E2 levels in the circulation. Cell Mol Immunol. 12(5):650-2.